1、 黄立锋,姚咏明,龚平,等.严重烧伤患者T淋巴细胞免疫功能变化及其与患者预后的关系[J].创伤外科杂志,2010,12(4):310-313. 黄立锋,姚咏明,龚平,等.严重烧伤患者T淋巴细胞免疫功能变化及其与患者预后的关系[J].创伤外科杂志,2010,12(4):310-313.
2、 孙旗,丁纯蕾,范倩,等.脓毒症小鼠不同组织炎症因子及T细胞亚群动态变化[J].中国临床医学,2022,29(6):999-1005. 孙旗,丁纯蕾,范倩,等.脓毒症小鼠不同组织炎症因子及T细胞亚群动态变化[J].中国临床医学,2022,29(6):999-1005.
3、 SCHWACHA M G,HOLLAND L T,CHAUDRY I H,et al.Genetic variability in the immune-inflammatory response after major burn injury[J].Shock,2005,23(2):123-128. SCHWACHA M G,HOLLAND L T,CHAUDRY I H,et al.Genetic variability in the immune-inflammatory response after major burn injury[J].Shock,2005,23(2):123-128.
4、 SACKS D,BAXTER B,CAMPBELL B C V,et al.Multisociety consensus quality improvement revised consensus statement for endovascular therapy of acute ischemic stroke[J].Int J Stroke,2018,13(6):612-632. SACKS D,BAXTER B,CAMPBELL B C V,et al.Multisociety consensus quality improvement revised consensus statement for endovascular therapy of acute ischemic stroke[J].Int J Stroke,2018,13(6):612-632.
5、 LUCK M E,LI X,HERRNREITER C J,et al.Ethanol intoxication and burn injury increases intestinal regulatory T cell population and regulatory T cell suppressive capability[J].Shock,2022,57(2):230-237. LUCK M E,LI X,HERRNREITER C J,et al.Ethanol intoxication and burn injury increases intestinal regulatory T cell population and regulatory T cell suppressive capability[J].Shock,2022,57(2):230-237.
6、 RANI M,ZHANG Q,SCHWACHA M G.Burn wound γδ T-cells support a Th2 and Th17 immune response[J].J Burn Care Res,2014,35(1):46-53. RANI M,ZHANG Q,SCHWACHA M G.Burn wound γδ T-cells support a Th2 and Th17 immune response[J].J Burn Care Res,2014,35(1):46-53.
7、 SASAKI J R,ZHANG Q,SCHWACHA M G.Burn induces a Th-17 inflammatory response at the injury site[J].Burns,2011,37(4):646-651. SASAKI J R,ZHANG Q,SCHWACHA M G.Burn induces a Th-17 inflammatory response at the injury site[J].Burns,2011,37(4):646-651.
8、 MIOSSEC P,KORN T,KUCHROO V K.Interleukin-17 and type 17 helper T cells[J].N Engl J Med,2009,361(9):888-898. MIOSSEC P,KORN T,KUCHROO V K.Interleukin-17 and type 17 helper T cells[J].N Engl J Med,2009,361(9):888-898.
9、 RENDON J L, CHOUDHRY M A.Th17 cells:critical mediators of host responses to burn injury and sepsis[J].J Leukoc Biol,2012,92(3):529-538. RENDON J L, CHOUDHRY M A.Th17 cells:critical mediators of host responses to burn injury and sepsis[J].J Leukoc Biol,2012,92(3):529-538.
10、 DEHNE M G,SABLOTZKI A,HOFFMANN A,et al.Alterations of acute phase reaction and cytokine production in patients following severe burn injury[J].Burns,2002,28(6):535-542. DEHNE M G,SABLOTZKI A,HOFFMANN A,et al.Alterations of acute phase reaction and cytokine production in patients following severe burn injury[J].Burns,2002,28(6):535-542.
11、 D’ALESSIO F R,KURZHAGEN J T,RABB H.Reparative T lymphocytes in organ injury[J].J Clin Invest,2019,129(7):2608-2618. D’ALESSIO F R,KURZHAGEN J T,RABB H.Reparative T lymphocytes in organ injury[J].J Clin Invest,2019,129(7):2608-2618.
12、 CLARK A,IMRAN J,MADNI T,et al.Nutrition and metabolism in burn patients[J].Burns Trauma,2017(5):11. CLARK A,IMRAN J,MADNI T,et al.Nutrition and metabolism in burn patients[J].Burns Trauma,2017(5):11.
13、 SCHWACHA M G,AYALA A,CHAUDRY I H.Insights into the role of gammadelta T lymphocytes in the immunopathogenic response to thermal injury[J].J Leukoc Biol,2000,67(5):644-650. SCHWACHA M G,AYALA A,CHAUDRY I H.Insights into the role of gammadelta T lymphocytes in the immunopathogenic response to thermal injury[J].J Leukoc Biol,2000,67(5):644-650.
14、 SCHWACHA M G.Gammadelta T-cells:Potential regulators of the post-burn inflammatory response[J].Burns,2009,35(3):318-326. SCHWACHA M G.Gammadelta T-cells:Potential regulators of the post-burn inflammatory response[J].Burns,2009,35(3):318-326.
15、 SCHWACHA M G,DANIEL T.Up-regulation of cell surface Toll-like receptors on circulating gammadelta T-cells following burn injury[J].Cytokine,2008,44(3):328-334. SCHWACHA M G,DANIEL T.Up-regulation of cell surface Toll-like receptors on circulating gammadelta T-cells following burn injury[J].Cytokine,2008,44(3):328-334.
16、 MULDER P P G,VLIG M,BOEKEMA B K H L,et al.Persistent systemic inflammation in patients with severe burn injury is accompanied by influx of immature neutrophils and shifts in T cell subsets and cytokine profiles[J].Front Immunol,2020(11):621222. MULDER P P G,VLIG M,BOEKEMA B K H L,et al.Persistent systemic inflammation in patients with severe burn injury is accompanied by influx of immature neutrophils and shifts in T cell subsets and cytokine profiles[J].Front Immunol,2020(11):621222.
17、 RAMIREZ K,WITHERDEN D A,HAVRAN W L.All hands on DE(T)C:Epithelial-resident γδ T cells respond to tissue injury[J].Cell Immunol,2015,296(1):57-61. RAMIREZ K,WITHERDEN D A,HAVRAN W L.All hands on DE(T)C:Epithelial-resident γδ T cells respond to tissue injury[J].Cell Immunol,2015,296(1):57-61.
18、 RANI M,ZHANG Q,SCHERER M R,et al.Activated skin γδ T-cells regulate T-cell infiltration of the wound site after burn[J].Innate Immun,2015,21(2):140-150. RANI M,ZHANG Q,SCHERER M R,et al.Activated skin γδ T-cells regulate T-cell infiltration of the wound site after burn[J].Innate Immun,2015,21(2):140-150.
19、 JIANG Y,YANG M,SUN X,et al.IL-10 NK and TGF-β NK cells play negative regulatory roles in HIV infection[J].BMC Infect Dis,2018,18(1):80. JIANG Y,YANG M,SUN X,et al.IL-10 NK and TGF-β NK cells play negative regulatory roles in HIV infection[J].BMC Infect Dis,2018,18(1):80.
20、 YANG H L,ZHOU W J,CHANG K K,et al.The crosstalk between endometrial stromal cells and macrophages impairs cytotoxicity of NK cells in endometriosis by secreting IL-10 and TGF-β[J].Reproduction,2017,154(6):815-825. YANG H L,ZHOU W J,CHANG K K,et al.The crosstalk between endometrial stromal cells and macrophages impairs cytotoxicity of NK cells in endometriosis by secreting IL-10 and TGF-β[J].Reproduction,2017,154(6):815-825.
21、 PERONA-WRIGHT G,MOHRS K,SZABA FM,et al.Systemic but not local infections elicit immunosuppressive IL-10 production by natural killer cells[J].Cell Host Microbe,2009,6(6):503-512. PERONA-WRIGHT G,MOHRS K,SZABA FM,et al.Systemic but not local infections elicit immunosuppressive IL-10 production by natural killer cells[J].Cell Host Microbe,2009,6(6):503-512.
22、 ELEMAM N M,RAMAKRISHNAN R K,HUNDT J E,et al.Innate lymphoid cells and natural killer cells in bacterial infections:Function,dysregulation,and therapeutic targets[J].Front Cell Infect Microbiol,2021(11):733564. ELEMAM N M,RAMAKRISHNAN R K,HUNDT J E,et al.Innate lymphoid cells and natural killer cells in bacterial infections:Function,dysregulation,and therapeutic targets[J].Front Cell Infect Microbiol,2021(11):733564.
23、 NOMURA T,KAWAMURA I,TSUCHIYA K,et al.Essential role of interleukin-12(IL-12)and IL-18 for gamma interferon production induced by listeriolysin O in mouse spleen cells[J].Infect Immun,2002,70(3):1049-1055. NOMURA T,KAWAMURA I,TSUCHIYA K,et al.Essential role of interleukin-12(IL-12)and IL-18 for gamma interferon production induced by listeriolysin O in mouse spleen cells[J].Infect Immun,2002,70(3):1049-1055.
24、 GUO Y,PATIL N K,LUAN L,et al.The biology of natural killer cells during sepsis[J].Immunology,2018,153(2):190-202. GUO Y,PATIL N K,LUAN L,et al.The biology of natural killer cells during sepsis[J].Immunology,2018,153(2):190-202.
25、 LIU M,LIANG S,ZHANG C.NK cells in autoimmune diseases:Protective or pathogenic?[J].Front Immunol,2021(12):624687. LIU M,LIANG S,ZHANG C.NK cells in autoimmune diseases:Protective or pathogenic?[J].Front Immunol,2021(12):624687.
26、 ZITTI B,BRYCESON Y T.Natural killer cells in inflammation and autoimmunity[J].Cytokine Growth Factor Rev,2018(42):37-46. ZITTI B,BRYCESON Y T.Natural killer cells in inflammation and autoimmunity[J].Cytokine Growth Factor Rev,2018(42):37-46.
27、 HIGHTON A J,SCHUSTER I S,DEGLI-ESPOSTI M A,et al.The role of natural killer cells in liver inflammation[J].Semin Immunopathol,2021,43(4):519-533. HIGHTON A J,SCHUSTER I S,DEGLI-ESPOSTI M A,et al.The role of natural killer cells in liver inflammation[J].Semin Immunopathol,2021,43(4):519-533.
28、 OSUKA A,HANSCHEN M,STOECKLEIN V,et al.A protective role for inflammasome activation following injury[J].Shock,2012,37(1):47-55. OSUKA A,HANSCHEN M,STOECKLEIN V,et al.A protective role for inflammasome activation following injury[J].Shock,2012,37(1):47-55.
29、 SCHNEIDER D F,GLENN C H,FAUNCE D E.Innate lymphocyte subsets and their immunoregulatory roles in burn injury and sepsis[J].J Burn Care Res,2007,28(3):365-379. SCHNEIDER D F,GLENN C H,FAUNCE D E.Innate lymphocyte subsets and their immunoregulatory roles in burn injury and sepsis[J].J Burn Care Res,2007,28(3):365-379.
30、 YADAVALLI G K,AULETTA J J,GOULD M P,et al.Deactivation of the innate cellular immune response following endotoxic and surgical injury[J].Exp Mol Pathol,2001,71(3):209-221. YADAVALLI G K,AULETTA J J,GOULD M P,et al.Deactivation of the innate cellular immune response following endotoxic and surgical injury[J].Exp Mol Pathol,2001,71(3):209-221.
31、 KAWASAKI T,KOBAYASHI M,HERNDON D N,et al.Role of natural killer dendritic cells in host resistance against pseudomonas aeruginosa infection after thermal injury in mice[J].Shock,2010,34(1):83-89. KAWASAKI T,KOBAYASHI M,HERNDON D N,et al.Role of natural killer dendritic cells in host resistance against pseudomonas aeruginosa infection after thermal injury in mice[J].Shock,2010,34(1):83-89.
32、 DEVINE R A,DILTZ Z,HALL M W,et al.The systemic immune response to pediatric thermal injury[J].Int J Burns Trauma,2018,8(1):6-16. DEVINE R A,DILTZ Z,HALL M W,et al.The systemic immune response to pediatric thermal injury[J].Int J Burns Trauma,2018,8(1):6-16.
33、 BENDER B S,WINCHURCH R A,THUPARI J N,et al.Depressed natural killer cell function in thermally injured adults:Successful in vivo and in vitro immunomodulation and the role of endotoxin[J].Clin Exp Immunol,1988,71(1):120-125. BENDER B S,WINCHURCH R A,THUPARI J N,et al.Depressed natural killer cell function in thermally injured adults:Successful in vivo and in vitro immunomodulation and the role of endotoxin[J].Clin Exp Immunol,1988,71(1):120-125.
34、 BLAZAR B A,RODRICK M L,O’MAHONY J B,et al.Suppression of natural killer-cell function in humans following thermal and traumatic injury[J].J Clin Immunol,1986,6(1):26-36. BLAZAR B A,RODRICK M L,O’MAHONY J B,et al.Suppression of natural killer-cell function in humans following thermal and traumatic injury[J].J Clin Immunol,1986,6(1):26-36.
35、 KHAWAR B,ABBASI M H,SHEIKH N.A panoramic spectrum of complex interplay between the immune system and IL-32 during pathogenesis of various systemic infections and inflammation[J].Euro J Med Res,2015,20(1):7. KHAWAR B,ABBASI M H,SHEIKH N.A panoramic spectrum of complex interplay between the immune system and IL-32 during pathogenesis of various systemic infections and inflammation[J].Euro J Med Res,2015,20(1):7.
36、 PAOLINI R,BERNARDINI G,MOLFETTA R,et al.NK cells and interferons[J].Cyto Grow Fac Rev,2015,26(2):113-120. PAOLINI R,BERNARDINI G,MOLFETTA R,et al.NK cells and interferons[J].Cyto Grow Fac Rev,2015,26(2):113-120.
37、 RAMíREZ-LABRADA A,PESINI C,SANTIAGO L,et al.All about(NK cell-mediated)death in two acts and an unexpected encore:Initiation,execution and activation of adaptive immunity[J].Front Immunol,2022(13):896228. RAMíREZ-LABRADA A,PESINI C,SANTIAGO L,et al.All about(NK cell-mediated)death in two acts and an unexpected encore:Initiation,execution and activation of adaptive immunity[J].Front Immunol,2022(13):896228.
38、 PRAGER I,WATZL C.Mechanisms of natural killer cell-mediated cellular cytotoxicity[J].J Leukoc Biol,2019,105(6):1319-1329. PRAGER I,WATZL C.Mechanisms of natural killer cell-mediated cellular cytotoxicity[J].J Leukoc Biol,2019,105(6):1319-1329.
39、 KUCUKSEZER U C,AKTAS CETIN E,ESEN F,et al.The role of natural killer cells in autoimmune diseases[J].Front Immunol,2021(12):622306. KUCUKSEZER U C,AKTAS CETIN E,ESEN F,et al.The role of natural killer cells in autoimmune diseases[J].Front Immunol,2021(12):622306.
40、 GHAZAWI F M,ZARGHAM R,GILARDINO M S,et al.Insights into the pathophysiology of hypertrophic scars and keloids:How do They differ?[J].Adv Skin Wound Care,2018,31(1):582-595. GHAZAWI F M,ZARGHAM R,GILARDINO M S,et al.Insights into the pathophysiology of hypertrophic scars and keloids:How do They differ?[J].Adv Skin Wound Care,2018,31(1):582-595.
41、 OSKERITZIAN C A.Mast cells and wound healing[J].Adv Wound Care,2012,1(1):23-28. OSKERITZIAN C A.Mast cells and wound healing[J].Adv Wound Care,2012,1(1):23-28.
42、 PARIHAR A,PARIHAR M S,MILNER S,et al.Oxidative stress and anti-oxidative mobilization in burn injury[J].Burns,2008,34(1):6-17. PARIHAR A,PARIHAR M S,MILNER S,et al.Oxidative stress and anti-oxidative mobilization in burn injury[J].Burns,2008,34(1):6-17.
43、 NIELSON C B,DUETHMAN N C,HOWARD J M,et al.Burns:Pathophysiology of systemic complications and current management[J].J Burn Care Res,2017,38(1):e469-e481. NIELSON C B,DUETHMAN N C,HOWARD J M,et al.Burns:Pathophysiology of systemic complications and current management[J].J Burn Care Res,2017,38(1):e469-e481.
44、 DOUAIHER J,SUCCAR J,LANCEROTTO L,et al.Development of mast cells and importance of their tryptase and chymase serine proteases in inflammation and wound healing[J].Adv Immunol,2014(122):211-252. DOUAIHER J,SUCCAR J,LANCEROTTO L,et al.Development of mast cells and importance of their tryptase and chymase serine proteases in inflammation and wound healing[J].Adv Immunol,2014(122):211-252.
45、 BANKOVA L G,LEZCANO C,PEJLER G,et al.Mouse mast cell proteases 4 and 5 mediate epidermal injury through disruption of tight junctions[J].J Immunol,2014,192(6):2812-2820. BANKOVA L G,LEZCANO C,PEJLER G,et al.Mouse mast cell proteases 4 and 5 mediate epidermal injury through disruption of tight junctions[J].J Immunol,2014,192(6):2812-2820.
46、 WILGUS T A,WULFF B C.The importance of mast cells in dermal scarring[J].Adv Wound Care,2014,3(4):356-365. WILGUS T A,WULFF B C.The importance of mast cells in dermal scarring[J].Adv Wound Care,2014,3(4):356-365.
47、 SANTOS F X,ARROYO C,GARCíA I,et al.Role of mast cells in the pathogenesis of postburn inflammatory response:reactive oxygen species as mast cell stimulators[J].Burns,2000,26(2):145-147. SANTOS F X,ARROYO C,GARCíA I,et al.Role of mast cells in the pathogenesis of postburn inflammatory response:reactive oxygen species as mast cell stimulators[J].Burns,2000,26(2):145-147.
48、 WILGUS T A,WULFF B C.The importance of mast cells in dermal scarring[J].Adv Wound Care,2014,3(4):356-365. WILGUS T A,WULFF B C.The importance of mast cells in dermal scarring[J].Adv Wound Care,2014,3(4):356-365.
49、 VENET M,BIDAR F,DERIVE M,et al.Persistently elevated soluble triggering receptor expressed on myeloid cells 1 and decreased monocyte human leucocyte antigen DR expression are associated with nosocomial infections in septic shock patients[J].Crit Care Explor,2023,5(3):e0869. VENET M,BIDAR F,DERIVE M,et al.Persistently elevated soluble triggering receptor expressed on myeloid cells 1 and decreased monocyte human leucocyte antigen DR expression are associated with nosocomial infections in septic shock patients[J].Crit Care Explor,2023,5(3):e0869.
50、 VENET F,TISSOT S,DEBARD A L,et al.Decreased monocyte human leukocyte antigen-DR expression after severe burn injury:Correlation with severity and secondary septic shock[J].Crit Care Med,2007,35(8):1910-1917. VENET F,TISSOT S,DEBARD A L,et al.Decreased monocyte human leukocyte antigen-DR expression after severe burn injury:Correlation with severity and secondary septic shock[J].Crit Care Med,2007,35(8):1910-1917.
51、 PERONNET E,BLEIN S,VENET F,et al.Immune profiling panel gene set identifies critically ill patients with low monocyte human leukocyte antigen-Dr expression:Preliminary results from the REAnimation low immune status marker(REALISM)study[J].Crit Care Med,2023,51(6):808-816. PERONNET E,BLEIN S,VENET F,et al.Immune profiling panel gene set identifies critically ill patients with low monocyte human leukocyte antigen-Dr expression:Preliminary results from the REAnimation low immune status marker(REALISM)study[J].Crit Care Med,2023,51(6):808-816.
52、 REDDELL L,COTTON B A.Antioxidants and micronutrient supplementation in trauma patients[J].Curr Opin Clin Nutr Metab Care,2012,15(2):181-187. REDDELL L,COTTON B A.Antioxidants and micronutrient supplementation in trauma patients[J].Curr Opin Clin Nutr Metab Care,2012,15(2):181-187.
53、 HORTON J W.Free radicals and lipid peroxidation mediated injury in burn trauma:the role of antioxidant therapy[J].Toxicology,2003,189(1-2):75-88. HORTON J W.Free radicals and lipid peroxidation mediated injury in burn trauma:the role of antioxidant therapy[J].Toxicology,2003,189(1-2):75-88.
54、 MA M Q,ZHENG S S,CHEN H L,et al.Protectin conjugates in tissue regeneration 1 inhibits macrophage pyroptosis by restricting NLRP3 inflammasome assembly to mitigate sepsis via the cAMP-PKA pathway[J].Lab Invest,2023,103(1):100028. MA M Q,ZHENG S S,CHEN H L,et al.Protectin conjugates in tissue regeneration 1 inhibits macrophage pyroptosis by restricting NLRP3 inflammasome assembly to mitigate sepsis via the cAMP-PKA pathway[J].Lab Invest,2023,103(1):100028.
55、 HORGAN A F,O’RIORDAIN D S,CHIN D H,et al.The role of cyclic adenosine monophosphate in the suppression of cellular immunity after thermal injury[J].Arch Surg,1994,129(12):1284-1289. HORGAN A F,O’RIORDAIN D S,CHIN D H,et al.The role of cyclic adenosine monophosphate in the suppression of cellular immunity after thermal injury[J].Arch Surg,1994,129(12):1284-1289.
56、 ZHANG L F,ZHANG X Y,WANG A C,et al.Bidirectional crosstalk of the cAMP/ROS-dependent signaling pathways in inflammatory macrophage:An activation of formononetin[J].Toxicol Appl Pharmacol,2023(472):116571. ZHANG L F,ZHANG X Y,WANG A C,et al.Bidirectional crosstalk of the cAMP/ROS-dependent signaling pathways in inflammatory macrophage:An activation of formononetin[J].Toxicol Appl Pharmacol,2023(472):116571.
57、 SCHWACHA M G.Macrophages and post-burn immune dysfunction[J].Burns,2003,29(1):1-14. SCHWACHA M G.Macrophages and post-burn immune dysfunction[J].Burns,2003,29(1):1-14.
58、 SERRA M B,BARROSO W A,DA SILVA N N,et al.From inflammation to current and alternative therapies involved in wound healing[J].Int J Inflam,2017(2017):3406215. SERRA M B,BARROSO W A,DA SILVA N N,et al.From inflammation to current and alternative therapies involved in wound healing[J].Int J Inflam,2017(2017):3406215.
59、 SUN J,LIAO Z,LI Z,et al.Down-regulation miR-146a-5p in Schwann cell-derived exosomes induced macrophage M1 polarization by impairing the inhibition on TRAF6/NF-κB pathway after peripheral nerve injury[J].Exp Neurol,2023(362):114295. SUN J,LIAO Z,LI Z,et al.Down-regulation miR-146a-5p in Schwann cell-derived exosomes induced macrophage M1 polarization by impairing the inhibition on TRAF6/NF-κB pathway after peripheral nerve injury[J].Exp Neurol,2023(362):114295.
60、 BLEARS E,SOMMERHALDER C,TOLIVER-KINSKY T,et al.Current problems in burn immunology[J].Cur Probl Surg,2020,57(6):100779. BLEARS E,SOMMERHALDER C,TOLIVER-KINSKY T,et al.Current problems in burn immunology[J].Cur Probl Surg,2020,57(6):100779.
61、 OGLE M E,SEGAR C E,SRIDHAR S,et al.Monocytes and macrophages in tissue repair:Implications for immunoregenerative biomaterial design[J].Exp Bio Med,2016,241(10):1084-1097. OGLE M E,SEGAR C E,SRIDHAR S,et al.Monocytes and macrophages in tissue repair:Implications for immunoregenerative biomaterial design[J].Exp Bio Med,2016,241(10):1084-1097.
62、 OLINGY C E,SAN EMETERIO C L,OGLE M E,et al.Non-classical monocytes are biased progenitors of wound healing macrophages during soft tissue injury[J].Sci Rep,2017,7(1):447. OLINGY C E,SAN EMETERIO C L,OGLE M E,et al.Non-classical monocytes are biased progenitors of wound healing macrophages during soft tissue injury[J].Sci Rep,2017,7(1):447.
63、 RODRIGUES M,KOSARIC N,BONHAM C A,et al.Wound healing:A cellular perspective[J].Physiol Rev,2019,99(1):665-706. RODRIGUES M,KOSARIC N,BONHAM C A,et al.Wound healing:A cellular perspective[J].Physiol Rev,2019,99(1):665-706.
64、 REHOU S,SHAHROKHI S,NATANSON R,et al.Antioxidant and trace element supplementation reduce the inflammatory response in critically ill burn patients[J].J Burn Care Res,2018,39(1):1-9. REHOU S,SHAHROKHI S,NATANSON R,et al.Antioxidant and trace element supplementation reduce the inflammatory response in critically ill burn patients[J].J Burn Care Res,2018,39(1):1-9.
65、 STRUDWICK X L,ADAMS D H,PYNE N T,et al.Systemic delivery of anti-integrin αl antibodies reduces early macrophage recruitment,inflammation,and scar formation in murine burn wounds[J].Adv Wound Care,2020,9(12):637-648. STRUDWICK X L,ADAMS D H,PYNE N T,et al.Systemic delivery of anti-integrin αl antibodies reduces early macrophage recruitment,inflammation,and scar formation in murine burn wounds[J].Adv Wound Care,2020,9(12):637-648.
66、 BERGQUIST M,H?STBACKA J,GLAUMANN C,et al.The time-course of the inflammatory response to major burn injury and its relation to organ failure and outcome[J].Burns,2019,45(2):354-363. BERGQUIST M,H?STBACKA J,GLAUMANN C,et al.The time-course of the inflammatory response to major burn injury and its relation to organ failure and outcome[J].Burns,2019,45(2):354-363.
67、 LOCATI M,CURTALE G,MANTOVANI A.Diversity,mechanisms,and significance of macrophage plasticity[J].Annu Rev Pathol,2020(15):123-147. LOCATI M,CURTALE G,MANTOVANI A.Diversity,mechanisms,and significance of macrophage plasticity[J].Annu Rev Pathol,2020(15):123-147.
68、 STIEGLITZ D,SCHMID T,CHHABRA N F,et al.TNF and regulatory T cells are critical for sepsis-induced suppression of T cells[J].Immun Inflam Dis,2015,3(4):374-385. STIEGLITZ D,SCHMID T,CHHABRA N F,et al.TNF and regulatory T cells are critical for sepsis-induced suppression of T cells[J].Immun Inflam Dis,2015,3(4):374-385.
69、 NAVEENKUMAR S K,HEMSHEKHAR M,JAGADISH S,et al.Melatonin restores neutrophil functions and prevents apoptosis amid dysfunctional glutathione redox system[J].J Pineal Res,2020,69(3):e12676. NAVEENKUMAR S K,HEMSHEKHAR M,JAGADISH S,et al.Melatonin restores neutrophil functions and prevents apoptosis amid dysfunctional glutathione redox system[J].J Pineal Res,2020,69(3):e12676.
70、 LI Y,TAN R,LI R,et al.PKM2/STAT1-mediated PD-L1 upregulation on neutrophils during sepsis promotes neutrophil organ accumulation by serving an anti-apoptotic role[J].J Inflam(Lond),2023,20(1):16. LI Y,TAN R,LI R,et al.PKM2/STAT1-mediated PD-L1 upregulation on neutrophils during sepsis promotes neutrophil organ accumulation by serving an anti-apoptotic role[J].J Inflam(Lond),2023,20(1):16.
71、 HUANG J,ZHU Z,JI D,et al.Single-cell transcriptome profiling reveals neutrophil heterogeneity and functional multiplicity in the early stage of severe burn patients[J].Front Immunol,2021(12):792122. HUANG J,ZHU Z,JI D,et al.Single-cell transcriptome profiling reveals neutrophil heterogeneity and functional multiplicity in the early stage of severe burn patients[J].Front Immunol,2021(12):792122.
72、 BRUSERUD ?,MOSEVOLL K A,BRUSERUD ?,et al.The regulation of neutrophil migration in patients with sepsis:the complexity of the molecular mechanisms and their modulation in sepsis and the heterogeneity of sepsis patients[J].Cells,2023,12(7):1003. BRUSERUD ?,MOSEVOLL K A,BRUSERUD ?,et al.The regulation of neutrophil migration in patients with sepsis:the complexity of the molecular mechanisms and their modulation in sepsis and the heterogeneity of sepsis patients[J].Cells,2023,12(7):1003.
73、 YANG Y,LIU L,GUO Z,et al.Investigation and assessment of neutrophil dysfunction early after severe burn injury[J].Burns,2021,47(8):1851-1862. YANG Y,LIU L,GUO Z,et al.Investigation and assessment of neutrophil dysfunction early after severe burn injury[J].Burns,2021,47(8):1851-1862.
74、 LAGGNER M,LINGITZ M T,COPIC D,et al.Severity of thermal burn injury is associated with systemic neutrophil activation[J].Sci Rep,2022,12(1):1654. LAGGNER M,LINGITZ M T,COPIC D,et al.Severity of thermal burn injury is associated with systemic neutrophil activation[J].Sci Rep,2022,12(1):1654.
75、 HAMPSON P,DINSDALE R J,WEARN C M,et al.Neutrophil dysfunction,immature granulocytes,and cell-free DNA are early biomarkers of sepsis in burn-injured patients:A prospective observational cohort study[J].Ann Surg,2017,265(6):1241-1249. HAMPSON P,DINSDALE R J,WEARN C M,et al.Neutrophil dysfunction,immature granulocytes,and cell-free DNA are early biomarkers of sepsis in burn-injured patients:A prospective observational cohort study[J].Ann Surg,2017,265(6):1241-1249.
76、 MULDER P P G,KOENEN H J P M,VLIG M,et al.Burn-induced local and systemic immune response:Systematic review and meta-analysis of animal studies[J].J Invest Dermatol,2022,142(11):3093-3109.e15. MULDER P P G,KOENEN H J P M,VLIG M,et al.Burn-induced local and systemic immune response:Systematic review and meta-analysis of animal studies[J].J Invest Dermatol,2022,142(11):3093-3109.e15.
77、 MULDER P P G,VLIG M,FASSE E,et al.Burn-injured skin is marked by a prolonged local acute inflammatory response of innate immune cells and pro-inflammatory cytokines[J].Front Immunol,2022(13):1034420. MULDER P P G,VLIG M,FASSE E,et al.Burn-injured skin is marked by a prolonged local acute inflammatory response of innate immune cells and pro-inflammatory cytokines[J].Front Immunol,2022(13):1034420.
78、 MORTAZ E,ALIPOOR S D,ADCOCK I M,et al.Update on neutrophil function in severe inflammation[J].Front Immunol,2018(9):2171. MORTAZ E,ALIPOOR S D,ADCOCK I M,et al.Update on neutrophil function in severe inflammation[J].Front Immunol,2018(9):2171.
79、 MULDER P P G,VLIG M,BOEKEMA B K H L,et al.Persistent systemic inflammation in patients with severe burn injury is accompanied by influx of immature neutrophils and shifts in T cell subsets and cytokine profiles[J].Front Immunol,2021(11):621222. MULDER P P G,VLIG M,BOEKEMA B K H L,et al.Persistent systemic inflammation in patients with severe burn injury is accompanied by influx of immature neutrophils and shifts in T cell subsets and cytokine profiles[J].Front Immunol,2021(11):621222.
80、 RAVINDRAN M,KHAN M A,PALANIYAR N.Neutrophil extracellular trap formation:physiology,pathology,and pharmacology[J].Biomolecules,2019,9(8):365. RAVINDRAN M,KHAN M A,PALANIYAR N.Neutrophil extracellular trap formation:physiology,pathology,and pharmacology[J].Biomolecules,2019,9(8):365.
81、 UHL B,VADLAU Y,ZUCHTRIEGEL G,et al.Aged neutrophils contribute to the first line of defense in the acute inflammatory response[J].Blood,2016,128(19):2327-2337. UHL B,VADLAU Y,ZUCHTRIEGEL G,et al.Aged neutrophils contribute to the first line of defense in the acute inflammatory response[J].Blood,2016,128(19):2327-2337.
82、 ZHANG P,ZOU B,LIOU Y C,et al.The pathogenesis and diagnosis of sepsis post burn injury[J].Burns Trauma,2021(9):tkaa047. ZHANG P,ZOU B,LIOU Y C,et al.The pathogenesis and diagnosis of sepsis post burn injury[J].Burns Trauma,2021(9):tkaa047.
83、 JESCHKE M G,van BAAR M E,CHOUDHRY M A,et al.Burn injury[J].Nat Rev Dis Primers,2020,6(1):11. JESCHKE M G,van BAAR M E,CHOUDHRY M A,et al.Burn injury[J].Nat Rev Dis Primers,2020,6(1):11.
84、 STANOJCIC M,ABDULLAHI A,REHOU S,et al.Pathophysiological response to burn injury in adults[J].Ann surg,2018,267(3):576-584. STANOJCIC M,ABDULLAHI A,REHOU S,et al.Pathophysiological response to burn injury in adults[J].Ann surg,2018,267(3):576-584.
85、 JESCHKE M G,HERNDON D N.Burns in children:standard and new treatments[J].Lancet,2014,383(9923):1168-1178. JESCHKE M G,HERNDON D N.Burns in children:standard and new treatments[J].Lancet,2014,383(9923):1168-1178.